As we all know, the Clinton Foundation was all over Haiti when the earthquake happened and that people of course went missing and rumors of organ harvesting abound.
I just watched a video in regards to Houston and what might be happening there due to Harvey's destruction. I don't want to go into the video too much because it would appear to be too loosely related to PG, but here's how it's related:
As you're watching the video that's linked at the end of this thread, please consider
1) organ harvesting
2) people shuttled to places of containment
3) missing people
4) blood drives/donations/type casting
5) what we know happens to cops, investigators, and others who aren't in "the club"
6) schizophrenia and how it's related to adrenochrome
1.Adrenochrome is formed in the body, but we do not know whether more is formed in schizophrenics or where it is made, or are schizophrenics more suscep-tible to the effects of the aminochromes because they lack antioxidants or other protective factors. Future research will set-tle these issues. We need to know where the transformation occurs, in what quan-tities, and why. We need to know the endproducts of catecholamine oxidation and their pathways. We need to know which enzymes are involved and/or dam-aged, and we need more effective blocking agents which are effective in smaller doses but have the safety, and efficacy of vitamins.
Adrenolutin is another derivative of adrenochrome and more stable in blood. Hoffer and Kenyon (1957) showed that the compound made in blood from adrenaline (Leach and Heath, 1956), was adrenolutin. Leach and Heath found that the rate of con-version was greater in schizophrenic blood compared to normal blood.
I know I may seem like I'm going way out in left field, but hang with me just a bit longer, and consider how many homeless people are alcoholics. Also think about how PG researchers believe that adrenochrome is one of the major factors at play with SRA.
Only three of the aminochromes have been tested for hallucinogenic properties: adrenochrome, adrenolutin and leuko-adrenochrome (5.6 dihydroxy N Methyl in-dole). The first two are hallucinogens but the third is not. On the contrary, it is a very effective anti-anxiety compound when given sublingually for some very tense in-dividuals, producing an effect within five to ten minutes. The evidence for these con-clusions is available in The Hallucinogens (Hoffer, Osmond, 1967; Hoffer, 1962). The reactions induced by adrenochrome and by adrenolutin are somewhat different. Adrenolutin causes changes which are more subtle, with fewer perceptual illusions. It tends to flatten mood more and its effect lasts longer. Adrenochrome causes more perceptual changes but they are rarely as pronounced as those caused by LSD or mescalin. Its effect may last a long time. It produced a two-week paranoid depression in one of us (A.H.) and a one-week paranoid depressive reaction with visual illusions in a distinguished colleague of ours.
Adrenochrome potentiated the activity of LSD in a few alcoholics.
Between 1954 and 1962 we treated several thousand alcoholics with psychedelic therapy using LSD, usually 200 to 400 mcg. In the usual reaction the first changes would occur in about one hour. Increased anxiety was usually the first reaction. Within two hours they would ex-perience the usual reaction. Anxiety would fluctuate but was seldom high or a prob-lem. However, many alcoholics did not have the usual reaction even with 400 mcg. They remained very tense and uncomfortable all the time. This group did not profit from their experience. In a few patients an in-jection of adrenochrome after two hours would, within a few minutes, bring on the typical LSD reaction. We concluded that LSD did not act as an hallucinogen per se but that it induced an increase in the pro-duction of adrenochrome which was the hallucinogen. An individual who could not make enough adrenochrome would not be able to have the typical LSD reaction. This conclusion was supported by our earlier finding that vitamin B3 markedly reduced the intensity of LSD reactions whether given before or during the LSD reaction. By blocking the adrenochrome effect it would also block the effect of LSD. It would also explain why Brom LSD, a very potent antiserotonin, would not be an hallucino-gen. Brom LSD probably has no effect on adrenalin oxidation and would not increase the formation of adrenochrome. These are interesting speculations. Perhaps now with increasing interest in free radical hypoth-eses and in oxidized derivatives of the catecholamines, scientists will direct their interest back into these areas.
Maybe, in order to see the entire evil picture, we need to expand our manner of thinking and consider how PG involves much more than the children. While the kids are of course the main source of our concerns, the PG spider's web is vast and wide.
For those who do not believe the adrenochrome angle of PG, consider the following taken from the same webpage as the above info:
Adrenochrome and adrenolutin are hallucinogenic. We need more information about the other aminochromes. We need to know which synapses are inhibited, which receptors are involved.
Inhibiting the formulation of adreno-chrome is therapeutic. We need to know why the orthomolecular approach works.
That the adrenochrome hypothesis is not destroyed does not prove it is correct. Time will tell. It does prove it is a good, testable hypothesis and will continue to direct research in future as it has in the past. Why, then, has it been ignored for nearly thirty years?
We may be too close to the subject to really understand the reasons why the adrenochrome hypothesis has been ignored for so long. It did receive a certain amount of notoriety at the beginning, but it was quickly shot down by American psychia-trists led by the National Institute of Men-tal Health. A medical historian may one day be able to examine the issues more capa-bly. In our opinion, there were two main classes of opinion: scientific, and political. The political opposition prevented any se-rious examination of the consequences of the adrenochrome hypothesis. Inadequate as it then was, it was able to direct our re-search in Saskatchewan for 15 years, and could have been examined much more quickly and thoroughly by research insti-tutes which grew very quickly in the U.S.A., beginning in 1955.
Link to adrenochrome info quoted above: http://orthomolecular.org/library/jom/1999/articles/1999-v14n01-p049.shtml, http://archive.is/APvcD
Please watch the following 10 minute video while considering everything I mentioned above and all that we believe PG encompasses.