Profile overview for ivegotaqueso.
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This user has mostly submitted to the following subverses (showing top 5):

1 submissions to fatpeoplehate

This user has so far shared a total of 1 links, started a total of 0 discussions and submitted a total of 602 comments.

Voting habits

Submissions: This user has upvoted 192 and downvoted 17 submissions.

Comments: This user has upvoted 561 and downvoted 88 comments.

Submission ratings

5 highest rated submissions:

5 lowest rated submissions:

Comment ratings

3 highest rated comments:

/r/punchablefaces now fully controlled by the SRS cult submitted by smk to MeanwhileOnReddit

ivegotaqueso 0 points 58 points (+58|-0) ago

From the Hot page of punchablefaces, I was looking at a Tess thread full of shitlord comments right as it started getting wonky. I have some screenshots of the moderation team changing right before my eyes....(notice how there were first only 3 mods....then an ArchangelGab showed up...then 12 entirely new mods were there...or something like that).

https://slimgur.com/images/2015/08/10/b2fa5c1ff0293f3197a8522ef58cec01.png

https://slimgur.com/images/2015/08/10/b7f7079a2e5ada1cb9ca1ac5fd2d9ace.png

https://slimgur.com/images/2015/08/10/fe061ee65c5ae6945eb28f04289f9ee0.png

Edit: Of the first 3 mods in the first screenshot, all 3 were new accounts with 0 activity...4 hours previously, one of the new mods posted a "this sub is dead" thread

deleted by user submitted by deleted to fatpeoplehate

ivegotaqueso 0 points 45 points (+45|-0) ago

You are still young. Actually if you were my kid, I'd disapprove of marriage before the age of 25...but that's just me. It's great you haven't had children yet I presume. Divorce is an option.

Also try couples counseling but by the sound of it he's resigned to being a fatty. He's not going to lose weight unless he wants to lose weight.

Dating sites should require FPH-style verification if you select 'athletic' or 'thin' for body type submitted by Silverstaryu to fatpeoplehate

ivegotaqueso 0 points 45 points (+45|-0) ago

You forget that to fat people, athletic = goes outdoors but not for food.

I'm fucking serious.

3 lowest rated comments:

Oh guess what's new. Boogie2988 is crying about his feelings again. submitted by DelusionalHominids to fatpeoplehate

ivegotaqueso 2 points -1 points (+1|-2) ago

lack of rigorous scientific data

You're aware that this simply means that more research needs to be done? Obesity was not an issue 20 years ago. It has only recently become an area of interest/research in areas like psychology, ~10 years ago when people started getting noticeably fatter in the US.

I did state that this area is gaining in popularity as obesity grows to become a bigger and bigger problem, especially in developed nations.

A lot of research is also relatively new, some new tools are being developed as well to help science explore this area of research:

For example: http://www.sciencedirect.com/science/article/pii/S0195666308006223

Previous research has found similarities between addiction to psychoactive substances and excessive food consumption. Further exploration is needed to evaluate the concept of “food addiction,” as there is currently a lack of psychometrically validated measurement tools in this area. The current study represents a preliminary exploration of the Yale Food Addiction Scale (YFAS), designed to identify those exhibiting signs of addiction towards certain types of foods (e.g., high fat and high sugar). Survey data were collected from 353 respondents from a stratified random sample of young adults. In addition to the YFAS, the survey assessed eating pathology, alcohol consumption and other health behaviors. The YFAS exhibited adequate internal reliability, and showed good convergent validity with measures of similar constructs and good discriminant validity relative to related but dissimilar constructs. Additionally, the YFAS predicted binge-eating behavior above and beyond existing measures of eating pathology, demonstrating incremental validity. The YFAS is a sound tool for identifying eating patterns that are similar to behaviors seen in classic areas of addiction. Further evaluation of the scale is needed, especially due to a low response rate of 24.5% and a non-clinical sample, but confirmation of the reliability and validity of the scale has the potential to facilitate empirical research on the concept of “food addiction”.

That is just one example, as recent as 2009, that shows how tools are still being developed to evaluate issues like food addiction.

Support for the food addiction hypothesis comes from alterations in neurochemistry (dopamine, endogenous opioids), neuroanatomy (limbic system), and self-medication behaviors. Foods identified as having potential addictive properties include sweets, carbohydrates, fats, sweet/fat combinations, and possibly processed and/or high salt foods.

Actually this supports what I have said.

Support for the food addiction hypothesis

Which means that these things support their food addiction hypothesis, they do not negate it.

Also if you want to see more (this is just ONE example):

archpsyc.jamanetwork.com/article.aspx?articleid=1107239&resultclick=1

" Neural Correlates of Food Addiction "

Results Food addiction scores (N = 39) correlated with greater activation in the anterior cingulate cortex, medial orbitofrontal cortex, and amygdala in response to anticipated receipt of food (P < .05, false discovery rate corrected for multiple comparisons in small volumes). Participants with higher (n = 15) vs lower (n = 11) food addiction scores showed greater activation in the dorsolateral prefrontal cortex and the caudate in response to anticipated receipt of food but less activation in the lateral orbitofrontal cortex in response to receipt of food (false discovery rate–corrected P < .05).

Conclusions Similar patterns of neural activation are implicated in addictive-like eating behavior and substance dependence: elevated activation in reward circuitry in response to food cues and reduced activation of inhibitory regions in response to food intake.

One-third of American adults are now obese1 and obesity-related disease is the second leading cause of preventable death.2 Unfortunately, most obesity treatments do not result in lasting weight loss because most patients regain their lost weight within 5 years.3

Based on numerous parallels in neural functioning associated with substance dependence (in the current article, the terms substance dependence and addiction are used interchangeably to represent a diagnosis of substance dependence as defined by the DSM-IV-TR4) and obesity, theorists have proposed that addictive processes may be involved in the etiology of obesity.5,6 Food and drug use both result in dopamine release in mesolimbic regions and the degree of release correlates with subjective reward from both food and drug use.6,7 **Similar patterns of brain activation in response to food and drug cues have also been found. Individuals with vs without substance dependence show greater activation in brain regions that encode the reward value of stimuli **(eg, the orbitofrontal cortex [OFC], amygdala, insula, striatum, anterior cingulate cortex [ACC], and dorsolateral prefrontal cortex [DLPFC])8,9 and greater dopamine release in the dorsal striatum in response to drug cues.10 Similarly, obese vs lean individuals show greater activation in the OFC, amygdala, ACC, striatum, and mediodorsal thalamus in response to food cues8 and greater activation in regions associated with cue-related craving for drugs, such as the ACC, striatum, insula, and the DLPFC, in response to anticipated receipt of palatable foods.11- 13

Although obese and substance-dependent individuals show hyperresponsivity of reward learning regions to food and substance cues, respectively, actual intake of food and drugs is associated with reduced reward circuitry activation. Obese vs lean individuals show less dorsal striatal and medial OFC activation in response to palatable food intake,14,15 echoing evidence that substance-dependent individuals exhibit blunted dopaminergic release during actual drug consumption and report weaker subjective reward relative to healthy controls.16- 19 Results dovetail with evidence of reduced D2 receptor availability in obese and substance-dependent individuals vs healthy controls.20,21 These findings have prompted the theory that individuals who experience less reward from food intake may overeat to compensate for this reward deficit.20,22

Although there are strong parallels in brain regions that encode reward from drugs and palatable foods and in neural abnormalities associated with substance dependence and obesity, these findings may tell us little about true“food addiction” (FA). Obesity is strongly linked to excess food consumption, but other factors contribute to unhealthy weight gain, such as physical inactivity.23 Additionally, excess consumption is not necessarily indicative of substance dependence; whereas 40% of college students binge drink,24 only 6% meet criteria for alcohol dependence.25 Thus, to more directly assess FA, it would be useful to identify participants who may exhibit signs of dependence in their eating behavior. [I italicized these so that you will realize that this only means by "true food addiction" they mean that they were only able get samples from obese people, not people who are known to have an overt addiction to foodCurrently, a diagnosis of substance dependence is given when sufficient behavioral criteria are met (Table 1). The Yale Food Addiction Scale (YFAS) was developed to operationalize the construct of palatable food dependence based on the DSM-IV-TR4 substance dependence criteria.26 The identification of individuals who exhibit FA symptoms would allow for more direct examinations of the neurobiological similarities between substance dependence and compulsive food consumption.

EDIT: FUCK I hit reply one too many times...it wouldn't go because it was check my bits...so there are a lot of deleted posts below because it was submitting the same thing, and I deleted the repeats

Oh guess what's new. Boogie2988 is crying about his feelings again. submitted by DelusionalHominids to fatpeoplehate

ivegotaqueso 1 points -1 points (+0|-1) ago

It's an area of study and there is real neuroscience research being done behind it as we speak. I don't think you even read the article?

It's your call though to keep yourself ignorant. Just because you say you don't believe it because it doesn't fit your feelings on the subject, doesn't mean that these studies suddenly become invalidated. As shitlords I think this community prides itself on keeping informed and up to date on the science and research being done on obesity. Plugging your ears and pretending this research doesn't exist is basically what these fat lazy people are doing with health medicine. Don't be like these (mentally and physically) fat lazy people.

Oh guess what's new. Boogie2988 is crying about his feelings again. submitted by DelusionalHominids to fatpeoplehate

ivegotaqueso 1 points -1 points (+0|-1) ago

Just because something is being researched doesn't mean it's real. You don't understand science if you think otherwise.

This is just the most idiotic thing I've seen a shitlord post in FPH. As someone with a background in research and who actually have worked in a scientific setting, your ignorance about research and what a field of study entails is just overly, apparently, lacking in knowledge.

Just because someone disagrees with your beliefs doesn't mean they're a fat sympathizer (I use the word belief, because you are treating the subject of addiction as a belief rather than looking at the research out there; I bet you have no idea what something as basic as a neurotransmitter is, and what it's role in addiction entails?). It is a very childish attitude.

You're being very petulant and exude the equivalent of fat logic when it comes to scientific research. Again plugging your ears and pretending research in an area that is published in multiple peer reviewed journals by a panel of no-bullshit academics is "not real" is just ridiculous, and is the type of thinking stupid fat people fall into when an argument doesn't go their way.

Again it's your call to keep yourself ignorant, but that itself is no excuse to pretend that if science/research doesn't fit your perspective, then it is not "real."

Edit: also apparently you never heard me the first time:

I wouldn't have a good opinion of an addict. It can be used as an excuse, for sure, but that doesn't mean that an addict gets a free pass to fuck up. Even addicts should be shamed to change, especially if they are aware of their problems and know how to fix them but refuse to.

Fuck off with your bullshit man. You're beginning to sound like a fat tumblerina who wants to pretend that an obesity research finding is not "real" because of HAES.